CASE REPORT
Acute peripheral balance system impairment in the course of vestibular neuritis
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Klinika Otolaryngologii, Onkologii Laryngologicznej, Audiologii i Foniatrii, Uniwersytet Medyczny w Łodzi, Polska
Submission date: 2021-11-22
Acceptance date: 2022-03-07
Publication date: 2022-07-06
Corresponding author
Marzena Bielińska
Klinika Otolaryngologii, Onkologii Laryngologicznej, Audiologii i Foniatrii, Uniwersytet Medyczny w Łodzi, Stefana Żeromskiego 113, 90-549, Łódź, Polska
LW 2022;100(2):98-100
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Summary: The aim of the study was to present a case of acute peripheral damage to the balance system in the course of vestibular neuritis. 32 year-old female was admitted to the Department of Otolaryngology, Laryngological Oncology, Audiology and Phoniatrics – II Department of Otolaryngology, of Lodz Medical University, due to sudden onset of severe vertigo with a sensation of spinning of her surroundings, nausea and lasting several hours vomiting. She had never experienced such symptoms before. She also did not report any other ailments. She experienced no hearing disorders or tinnitus and she hadn’t been treated so far due to these ailments so far. ENT (ear, nose, throat) examination, audiological and otoneurological diagnostics, imaging (CT scans of the head) and laboratory tests were performed. The patient was diagnosed with acute peripheral damage to the balance system in the course of right-sided vestibular neuritis. Symptomatic treatment was delivered: piracetam, vinpocetine. Individually selected kinesiotherapy consisting of habituation exercises together with balance and coordination exercises (performed by the patient – if possible – several times a day), was recommended after reducing vegetative symptoms. Conclusions: The etiology of vestibular neuritis is not fully explained in most cases. The scientists advocate for viral theory, but the
disroders of vestibular nuclei blood supply is either emphasized. The clinical picture is usually similar and involves
sudden, rapid symptoms, resulting from the labyrinthine deficite, often with complete loss of its function.